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30:T6d19,

Hello, Dr. Sadangi

31:T150fb,Bioscope | Rachel Delaney | Synoptic view

Rachel Delaney

XX (Female)

57y

11/12/1968

155 lbs

Synoptic view

Summary and suggestions

Practice prompt

Overview

Rachel Delaney is a postmenopausal woman whose care currently centers on early dilated cardiomyopathy linked to a pathogenic TTN variant (EF improved from 47% to 50% on carvedilol/lisinopril) and cardiovascular risk reduction. Contributing metabolic factors include prediabetes with insulin resistance, hyperlipidemia (now at target on statin therapy), excess visceral adiposity, and APOE ε4/ε4 status. Additional active issues are osteopenia, vitamin D insufficiency, postmenopausal hormonal insufficiency, and persistent fatigue with non-restorative sleep.

Current Diagnoses and Issues

Diagnosis	Status	Notes
Dilated cardiomyopathy	Early phenotype, improving	Heterozygous pathogenic TTN truncating variant; EF 47% (Oct 2025) → 50% (Mar 2026) on therapy
Hyperlipidemia	Controlled on therapy	LDL persistently elevated through early 2025 ; LDL 58 mg/dL and ApoB 62 mg/dL at target as of Mar 2026
Hypertension	Controlled	Diagnosed ~2019; BP 122/76 mmHg Mar 2026 ; lisinopril uptitrated to 20 mg daily
Prediabetes	Active	HbA1c 5.9%, HOMA-IR 2.4
Excess body adiposity	Active	Body fat 36.4%, android/gynoid 1.04, waist-to-hip 0.88, VAT 108 cm²
Osteopenia	Active	DXA lumbar spine T-score −1.4, femoral neck −1.1
Vitamin D insufficiency	Active	25-OH vitamin D 28 ng/mL
Functional vitamin B12 deficiency	Resolved on supplementation	MMA 0.38 → 0.18 µmol/L, B12 342 → 824 pg/mL by Mar 2026
Postmenopausal hormonal insufficiency	Active	Estradiol 18 pg/mL, FSH 44.6 mIU/mL, free testosterone 0.9 pg/mL
Persistent fatigue with sleep disturbance	Active	Nighttime waking, non-restorative sleep, cognitive sluggishness

Current Medications

Medication	Dose & Frequency	Indication
Carvedilol	3.125 mg twice daily	Dilated cardiomyopathy
Lisinopril	20 mg once daily	Hypertension / cardiomyopathy
Rosuvastatin	15 mg once daily	Hyperlipidemia / cardiovascular risk grouped

Current Supplements

Supplement	Dose & Frequency	Indication
Vitamin D3 (cholecalciferol)	2000 IU once daily	Vitamin D insufficiency
Calcium citrate	500 mg twice daily	Bone health / osteopenia
Methylcobalamin (B12)	2000 µg once daily	Functional B12 deficiency
Methylfolate	400 µg once daily	Methylation / homocysteine support
Trimethylglycine (betaine)	500 mg twice daily	Homocysteine / methylation support
Multivitamin	Standard dose once daily	General supplementation

Key Findings

Finding	Significance
TTN truncating variant (c.59201_59202del, p.Pro19734fs), heterozygous	ClinVar Pathogenic/Likely Pathogenic; associated with dilated cardiomyopathy 1G (autosomal dominant). Consistent with the documented early cardiomyopathy phenotype
Cardiac function and NT-proBNP	EF 47% (Oct 2025) → 50% (Mar 2026) with stable LV dilation; NT-proBNP 186 → 112 pg/mL, indicating response to therapy
LDL / ApoB	LDL 58 mg/dL and ApoB 62 mg/dL at target as of Mar 2026, down from LDL 138 / ApoB 98 in Aug 2025
APOE ε4/ε4 genotype	Associated with increased late-onset Alzheimer's risk and higher LDL/total cholesterol; reinforces aggressive vascular risk management
HbA1c 5.9% / HOMA-IR 2.4	Prediabetic range with insulin resistance (Aug 2025; no update since)
Vitamin D 28 ng/mL	Insufficient (Aug 2025; no update since)

Suggestions

Category	Item	Rationale
Testing	Recheck HbA1c and HOMA-IR — no update since Aug 2025	Track glycemic status; prediabetes remains active
Testing	Recheck 25-OH vitamin D on current D3 2000 IU	Last value insufficient (28 ng/mL); confirm repletion
Testing	Continue serial echocardiography and NT-proBNP monitoring	TTN-related cardiomyopathy; guide titration
Supplements	Consider increasing vitamin D3 (e.g., toward 4000 IU daily) if repeat 25-OH D remains <30 ng/mL	Persistent insufficiency despite 2000 IU
Lifestyle	Reinforce weight/visceral-fat reduction and physical activity	Addresses adiposity, insulin resistance, and cardiovascular risk
Lifestyle	Note swallowing limitation — patient cannot swallow pills; favor liquid, dissolvable, patch, or injectable formulations	Adherence and safe administration grouped

Recent chat conversations

Body fat

36.4 %

BMI

24.6 kg/m²

Weight

155 lbs

WATCH

Physical resilience

NORMAL

Metabolic health

Cardiovascular health

Neurocognitive health

Mental health

Cancer risk

Immune health

Gut health

Renal health

Hepatic health

Women's health (menopausal)

Blood health

Nutritional health

Environmental health

Healthy longevity

32:Ta2a4,

Summary and suggestions

Practice prompt

Overview

Current Diagnoses and Issues

Diagnosis	Status	Notes
Dilated cardiomyopathy	Early phenotype, improving	Heterozygous pathogenic TTN truncating variant; EF 47% (Oct 2025) → 50% (Mar 2026) on therapy
Hyperlipidemia	Controlled on therapy	LDL persistently elevated through early 2025 ; LDL 58 mg/dL and ApoB 62 mg/dL at target as of Mar 2026
Hypertension	Controlled	Diagnosed ~2019; BP 122/76 mmHg Mar 2026 ; lisinopril uptitrated to 20 mg daily
Prediabetes	Active	HbA1c 5.9%, HOMA-IR 2.4
Excess body adiposity	Active	Body fat 36.4%, android/gynoid 1.04, waist-to-hip 0.88, VAT 108 cm²
Osteopenia	Active	DXA lumbar spine T-score −1.4, femoral neck −1.1
Vitamin D insufficiency	Active	25-OH vitamin D 28 ng/mL
Functional vitamin B12 deficiency	Resolved on supplementation	MMA 0.38 → 0.18 µmol/L, B12 342 → 824 pg/mL by Mar 2026
Postmenopausal hormonal insufficiency	Active	Estradiol 18 pg/mL, FSH 44.6 mIU/mL, free testosterone 0.9 pg/mL
Persistent fatigue with sleep disturbance	Active	Nighttime waking, non-restorative sleep, cognitive sluggishness

Current Medications

Medication	Dose & Frequency	Indication
Carvedilol	3.125 mg twice daily	Dilated cardiomyopathy
Lisinopril	20 mg once daily	Hypertension / cardiomyopathy
Rosuvastatin	15 mg once daily	Hyperlipidemia / cardiovascular risk grouped

Current Supplements

Supplement	Dose & Frequency	Indication
Vitamin D3 (cholecalciferol)	2000 IU once daily	Vitamin D insufficiency
Calcium citrate	500 mg twice daily	Bone health / osteopenia
Methylcobalamin (B12)	2000 µg once daily	Functional B12 deficiency
Methylfolate	400 µg once daily	Methylation / homocysteine support
Trimethylglycine (betaine)	500 mg twice daily	Homocysteine / methylation support
Multivitamin	Standard dose once daily	General supplementation

Key Findings

Finding	Significance
TTN truncating variant (c.59201_59202del, p.Pro19734fs), heterozygous	ClinVar Pathogenic/Likely Pathogenic; associated with dilated cardiomyopathy 1G (autosomal dominant). Consistent with the documented early cardiomyopathy phenotype
Cardiac function and NT-proBNP	EF 47% (Oct 2025) → 50% (Mar 2026) with stable LV dilation; NT-proBNP 186 → 112 pg/mL, indicating response to therapy
LDL / ApoB	LDL 58 mg/dL and ApoB 62 mg/dL at target as of Mar 2026, down from LDL 138 / ApoB 98 in Aug 2025
APOE ε4/ε4 genotype	Associated with increased late-onset Alzheimer's risk and higher LDL/total cholesterol; reinforces aggressive vascular risk management
HbA1c 5.9% / HOMA-IR 2.4	Prediabetic range with insulin resistance (Aug 2025; no update since)
Vitamin D 28 ng/mL	Insufficient (Aug 2025; no update since)

Suggestions

Category	Item	Rationale
Testing	Recheck HbA1c and HOMA-IR — no update since Aug 2025	Track glycemic status; prediabetes remains active
Testing	Recheck 25-OH vitamin D on current D3 2000 IU	Last value insufficient (28 ng/mL); confirm repletion
Testing	Continue serial echocardiography and NT-proBNP monitoring	TTN-related cardiomyopathy; guide titration
Supplements	Consider increasing vitamin D3 (e.g., toward 4000 IU daily) if repeat 25-OH D remains <30 ng/mL	Persistent insufficiency despite 2000 IU
Lifestyle	Reinforce weight/visceral-fat reduction and physical activity	Addresses adiposity, insulin resistance, and cardiovascular risk
Lifestyle	Note swallowing limitation — patient cannot swallow pills; favor liquid, dissolvable, patch, or injectable formulations	Adherence and safe administration grouped

Recent chat conversations

Body fat

36.4 %

BMI

24.6 kg/m²

Weight

155 lbs

33:Tcf9c,

Problem list

Total: 10

Last scanned Jul 9, 2026 · 9:43 PM

Bioscope curates a trusted list of active problems from the full record, not every mention ever ingested. Confident entries sit in Active; anything uncertain is flagged for your review. Scanning is manual and only pulls records new since the last scan.

Active

Problem	Details	Source
Dilated cardiomyopathy	Heterozygous pathogenic TTN truncating variant; echocardiogram (Oct 2025) shows EF 47% with mild LV dilation; early phenotypic expression confirmed.
Excess body adiposity	Total body fat 36.4% (overfat range for females 50–59); android fat 40.2%; android-to-gynoid ratio 1.04; elevated waist-to-hip ratio 0.88; borderline VAT 108 cm².	· edited
Functional vitamin B12 deficiency	MMA 1.38 µmol/L (elevated; reference <0.27); serum B12 342 pg/mL (low-normal); consistent with functional B12 deficiency.	· edited
Hyperlipidemia	LDL persistently elevated: 130 mg/dL (2022), 138 mg/dL (2023), 142 mg/dL (2024), 138 mg/dL (early 2025); condition persists despite therapy.
Hypertension	Diagnosed ~2019; on lisinopril 10 mg daily; BP 122/76 mmHg as of March 2026.
Osteopenia	Lumbar spine T-score −1.4; femoral neck T-score −1.1 on DXA; consistent with osteopenia at both sites.
Persistent fatigue with sleep disturbance	Progressive fatigue over several years; also includes sleep disturbance (waking 1–2× nightly, non-restorative), cognitive sluggishness, and exertional dyspnea.
Postmenopausal hormonal insufficiency	Estradiol 18 pg/mL, FSH 44.6 mIU/mL, free testosterone 0.9 pg/mL — consistent with postmenopausal hormonal insufficiency.
Prediabetes	HbA1c 5.9% (upper end of normal / prediabetes range); HOMA-IR 2.4 (mildly elevated); no formal diabetes diagnosis.
Vitamin D insufficiency	25-OH vitamin D 28 ng/mL (insufficiency range; normal ≥30 ng/mL).

Retired problems — never had, or since resolved. Kept for the record; reactivate to move one back onto the active list.

Palpitations

Brief, self-resolving palpitation episodes noted; being evaluated in context of TTN-related DCM.

Never had

Mild depressive symptoms

PHQ-9 score 6 (mild); no formal psychiatric diagnosis assigned; mood changes attributed to multifactorial causes including stress and sleep disruption.

Never had

Methylation pathway impairment

Multiple homozygous/heterozygous variants in AHCY, BHMT, MTRR, MTHFR, COMT, SUOX impairing homocysteine remethylation and methylation pathways.

Never had

34:T6724,

Genetic findings

Genetic findings represent all variants determined to have clinical relevance after filtering and evaluation against current evidence from curated clinical databases.

Summary

Rachel Delaney carries the APOE ε4/ε4 genotype (homozygous), defined by rs429358 (homozygous C) and rs7412 (homozygous reference C). This is the highest-risk APOE configuration for late-onset Alzheimer's disease and is also associated with altered lipid metabolism. At 57 years old, proactive management of modifiable cardiovascular and cognitive risk factors is particularly relevant.

Clinical Significance

The ε4/ε4 genotype confers substantially elevated risk for late-onset Alzheimer's disease compared to the common ε3/ε3 genotype — often cited in the range of 8–12× increased risk — and is associated with an earlier average age of onset. That said, this is a predisposition, not a diagnosis: a meaningful proportion of ε4/ε4 individuals never develop Alzheimer's disease, and many individuals without ε4 do. The ε4 allele is also linked to higher LDL and total cholesterol levels, making cardiovascular risk factor management especially important in this context. The patient's APOE-excluding polygenic risk score (PRS) for Alzheimer's would provide complementary genetic risk information beyond the APOE locus alone.

Out-of-Chat Clinical Considerations

Vascular risk factor optimization — given the dual relevance to both Alzheimer's risk and lipid metabolism, lipid panel review and blood pressure management are reasonable priorities to address.
Cognitive baseline — at age 57, establishing a formal cognitive baseline (e.g., structured neuropsychological testing) could be useful for longitudinal monitoring, particularly given the elevated genetic predisposition.
Lifestyle counseling — aerobic exercise, sleep optimization, and dietary modifications (Mediterranean or MIND diet) have the most robust evidence for modifiable risk reduction in APOE ε4 carriers and may be worth discussing.
Genetic counseling — given the magnitude of risk associated with ε4/ε4, a genetics or dementia specialist consultation may be worth offering, particularly if the patient has not yet had an informed discussion about the implications for herself and first-degree relatives (each child has a 50–100% chance of inheriting at least one ε4 allele depending on the other parent's genotype).
Family history — targeted inquiry into first-degree relatives with dementia or early cognitive decline would help contextualize the clinical picture.

Want me to dig deeper?

I can pull the patient's APOE-excluding Alzheimer's polygenic risk score (PRS) from her genomic data to see how her broader genetic background stacks up alongside the ε4/ε4 result — want me to run that?
I can check her most recent lipid panel (LDL, total cholesterol, triglycerides) given the ε4 association with dyslipidemia — want me to look that up?
I can search her record for any existing cognitive, neurocognitive, or dementia-related diagnoses or assessments — run that?
I can check for any current medications where APOE status or cognitive/cardiovascular risk would be relevant context — want me to?

37:T1d01,

Summary

A clinician-facing synthesis of this patient's microbiome findings and their clinical significance, generated from the assessment. Interpret alongside the full results and the patient's clinical context.

Alpha diversity shows cohort-typical richness (292 taxa, 50th percentile) but notable unevenness, with a Berger-Parker dominance index of 0.15 (88th percentile) driven by Bacteroides stercoris at 14.67%, indicating a skewed community structure.
Bacteroidota is high at 38.79% (95th percentile) with Bacillota low at 57.72% (10th percentile), reflecting a phylum-level composition that diverges from the reference cohort's typical ratio.
Multiple anaerobic genera typically associated with non-gut sites (Peptoniphilus, Finegoldia, Anaerococcus, Murdochiella, Porphyromonas, Corynebacterium) are detected at the 100th percentile, a pattern that is atypical and warrants clinical consideration.
Campylobacter concisus DNA is detected at 0.0432%, which is consistent with the patient's reported recent international travel and does not require intervention in the absence of GI symptoms.
Butyrate production potential is at the 57th percentile (typical band), consistent with strong representation of multiple butyrate-producing genera (Faecalibacterium, Roseburia, Agathobacter) despite low levels of some individual keystone species.
Vitamin B6 (pyridoxine) production potential is at the 4th percentile (low band) and vitamin K2 (menaquinone) production potential is absent, representing the most notable functional pathway findings .
The patient reports daily probiotic supplement use but no fermented food intake; the community composition does not show a prominent Lactobacillus signal, suggesting the supplement may not be contributing substantially to the observed profile.

38:T946,

Brain age

+0.2 yrs · Normal

57.3

predicted brain age

vs. actual age 57.1 — scores 0.2 years older than expected

57.3 (brain)

57.1 (actual)

Younger-looking

Older-looking

39:T43d,

calcium citrate
vitamin D3

3a:T5d5,

Polygenic risk scores

3b:T5a08,

Health Dimension Triage

Prioritized clinical review across Bioscope health dimensions (sorted worst-to-best attention level).

Health Dimension Evaluations

🟡3

Cardiovascular HealthSub-critical

Dilated cardiomyopathy (TTN pathogenic variant, EF 47% at diagnosis, improving to 50%) with CAC score 92 at the 75th–80th percentile for age/sex, established hypertension on dual therapy, and a strong family history of premature coronary artery disease collectively place her at elevated cardiovascular morbidity and mortality risk. NT-proBNP has improved from 186 to 112 pg/mL and BP is now controlled at 122/76 mmHg, preventing a Critical designation, but the underlying structural heart disease, moderate coronary atherosclerosis, and heart-failure polygenic risk at the 93.9th percentile maintain high ongoing risk.

Pertinent Findings

Dilated cardiomyopathy: TTN heterozygous truncating variant (rs752948913, pathogenic/likely pathogenic for autosomal-dominant DCM)
LVEF 47% at diagnosis (October 2025), improved to 50% by March 2026
NT-proBNP 186 pg/mL (September 2025) → 112 pg/mL (March 2026)
Hypertension on carvedilol 3.125 mg BID and lisinopril 20 mg daily; BP now 122/76 mmHg
CAC score 92 (LAD 54, LCX 23, RCA 15): 75th–80th percentile for age/sex — moderate atherosclerotic burden
Heart-failure polygenic risk score: 93.9th percentile
Family history: father with MI at age 62, paternal grandfather with unclear heart condition (died in his 60s)
LDL reduced to 58 mg/dL and ApoB to 62 mg/dL by March 2026 (previously 138/98 mg/dL in August 2025)
Exertional dyspnea with moderate activity (stairs, brisk walking)
No documented NYHA functional class; no heart failure hospitalization on record

Biomarker Trends

NT-proBNP

Date	Value	Reference Range
29 Sept 2025	186 pg/mL	<125 pg/mL
1 Mar 2026	112 pg/mL	<125 pg/mL

Left Ventricular Ejection Fraction

Date	Value	Reference Range
21 Oct 2025	47 %	≥55%
1 Mar 2026	50 %	≥55%

Blood Pressure Systolic

Date	Value	Reference Range
1 Aug 2021	132 mmHg	<120 mmHg
15 Aug 2022	132 mmHg	<120 mmHg
14 Aug 2023	136 mmHg	<120 mmHg
12 Aug 2024	140 mmHg	<120 mmHg
4 Aug 2025	136 mmHg	<120 mmHg
29 Sept 2025	134 mmHg	<120 mmHg
14 Oct 2025	128 mmHg	<120 mmHg
1 Mar 2026	122 mmHg	<120 mmHg

Blood Pressure Diastolic

Date	Value	Reference Range
1 Aug 2021	84 mmHg	<80 mmHg
15 Aug 2022	84 mmHg	<80 mmHg
14 Aug 2023	86 mmHg	<80 mmHg
12 Aug 2024	88 mmHg	<80 mmHg
4 Aug 2025	84 mmHg	<80 mmHg
29 Sept 2025	84 mmHg	<80 mmHg
14 Oct 2025	80 mmHg	<80 mmHg
1 Mar 2026	76 mmHg	<80 mmHg

🟡4

Metabolic HealthSub-critical

Prediabetes (HbA1c 5.9%, fasting glucose trending upward from 92 to 98 mg/dL over four years), active hyperlipidemia (LDL 138 mg/dL in August 2025 before treatment intensification), excess body adiposity (body fat 36.4%, visceral adipose tissue 108 cm² borderline elevated), and insulin resistance (HOMA-IR 2.4) represent a cluster of metabolic risk factors not yet managed with pharmacotherapy. LDL and ApoB have improved significantly with recent treatment, and BMI remains in the normal range (24.6), preventing a Critical rating.

Pertinent Findings

Prediabetes: HbA1c 5.9%, glucose trending 92→94→96→98 mg/dL (2022–2025)
HOMA-IR 2.4 — insulin resistance
Hyperlipidemia: LDL 138 mg/dL, ApoB 98 mg/dL (August 2025); improved to LDL 58, ApoB 62 mg/dL (March 2026) on treatment
Triglycerides 155 mg/dL, HDL 51 mg/dL (August 2025)
Body fat 36.4% by DXA (August 2025) — in overfat range
Visceral adipose tissue 108 cm² — borderline elevated
Android-to-gynoid fat ratio 1.04 — central adiposity pattern
BMI 24.6 — normal range, stable weight 152–155 lb (2022–2025)
No pharmacologic treatment for prediabetes or insulin resistance documented
Father with elevated cholesterol; brother on a statin

Biomarker Trends

LDL Cholesterol

Date	Value	Reference Range
1 Aug 2021	130 mg/dL	<100 mg/dL
15 Aug 2022	130 mg/dL	<100 mg/dL
14 Aug 2023	138 mg/dL	<100 mg/dL
12 Aug 2024	142 mg/dL	<100 mg/dL
4 Aug 2025	138 mg/dL	<100 mg/dL
1 Mar 2026	58 mg/dL	<100 mg/dL

HDL Cholesterol

Date	Value	Reference Range
15 Aug 2022	52 mg/dL	>50 mg/dL
14 Aug 2023	50 mg/dL	>50 mg/dL
12 Aug 2024	50 mg/dL	>50 mg/dL
4 Aug 2025	51 mg/dL	>50 mg/dL

Triglycerides

Date	Value	Reference Range
15 Aug 2022	150 mg/dL	<150 mg/dL
14 Aug 2023	150 mg/dL	<150 mg/dL
12 Aug 2024	160 mg/dL	<150 mg/dL
4 Aug 2025	155 mg/dL	<150 mg/dL

ApoB

Date	Value	Reference Range
4 Aug 2025	98 mg/dL	<80 mg/dL
1 Mar 2026	62 mg/dL	<80 mg/dL

Glucose

Date	Value	Reference Range
15 Aug 2022	92 mg/dL	70–99 mg/dL
14 Aug 2023	94 mg/dL	70–99 mg/dL
12 Aug 2024	96 mg/dL	70–99 mg/dL
4 Aug 2025	98 mg/dL	70–99 mg/dL

HbA1c

Date	Value	Reference Range
4 Aug 2025	5.9 %	<5.7%

BMI

Date	Value	Reference Range
15 Aug 2022	24.2 kg/m²	18.5–24.9 kg/m²
14 Aug 2023	24.3 kg/m²	18.5–24.9 kg/m²
12 Aug 2024	24.5 kg/m²	18.5–24.9 kg/m²
4 Aug 2025	24.6 kg/m²	18.5–24.9 kg/m²

🟢8

Renal HealthGood

Kidney function is stable and consistently normal over four years of follow-up — creatinine 0.82–0.84 mg/dL and eGFR >60 mL/min/1.73 m² with no proteinuria on urinalysis. Hypertension and prediabetes represent risk factors, but no CKD diagnosis or progressive renal impairment is documented.

Pertinent Findings

Creatinine stable 0.82–0.84 mg/dL (2022–2025)
eGFR >60 mL/min/1.73 m² at all time points (2022–2025)
BUN 14–15 mg/dL — consistently normal
Urine protein negative on urinalysis (2022–2025)
No urine albumin-to-creatinine ratio on record
No CKD diagnosis
Hypertension and prediabetes present as CKD risk factors
On lisinopril — provides renoprotection

Biomarker Trends

Creatinine

Date	Value	Reference Range
15 Aug 2022	0.82 mg/dL	0.5–1.1 mg/dL
14 Aug 2023	0.84 mg/dL	0.5–1.1 mg/dL
12 Aug 2024	0.83 mg/dL	0.5–1.1 mg/dL
4 Aug 2025	0.84 mg/dL	0.5–1.1 mg/dL

🟢8

Hepatic HealthGood

Liver enzymes (ALT, AST, alkaline phosphatase) and synthetic markers (albumin, total protein, bilirubin) are consistently within normal limits across all four annual panels (2022–2025), with no liver disease diagnosis, hepatomegaly, or liver imaging findings. No GGT data are available.

Pertinent Findings

ALT 18–21 U/L, AST 22–24 U/L — consistently normal (2022–2025)
Alkaline phosphatase 68–72 U/L — normal
Total bilirubin 0.6–0.7 mg/dL — normal
Albumin 4.2–4.4 g/dL — normal synthetic function
No liver disease diagnosis (no NAFLD/MASLD, hepatitis, cirrhosis)
No hepatomegaly on examination
GGT not measured
Alcohol use: ~3–5 drinks/week (within low-risk range)

Biomarker Trends

ALT

Date	Value	Reference Range
15 Aug 2022	18 U/L	7–40 U/L
14 Aug 2023	20 U/L	7–40 U/L
12 Aug 2024	19 U/L	7–40 U/L
4 Aug 2025	21 U/L	7–40 U/L

AST

Date	Value	Reference Range
15 Aug 2022	22 U/L	10–40 U/L
14 Aug 2023	24 U/L	10–40 U/L
12 Aug 2024	23 U/L	10–40 U/L
4 Aug 2025	24 U/L	10–40 U/L

⚪—

Neurocognitive HealthUnknown

No summary reasoning recorded.

Pertinent Findings

No findings recorded

Biomarker Trends

No biomarker trends recorded for this dimension

⚪—

Blood HealthUnknown

No summary reasoning recorded.

Pertinent Findings

No findings recorded

Biomarker Trends

No biomarker trends recorded for this dimension

⚪—

Immune HealthUnknown

No summary reasoning recorded.

Pertinent Findings

No findings recorded

Biomarker Trends

No biomarker trends recorded for this dimension

⚪—

Gut HealthUnknown

No summary reasoning recorded.

Pertinent Findings

No findings recorded

Biomarker Trends

No biomarker trends recorded for this dimension

⚪—

Cancer RiskUnknown

No summary reasoning recorded.

Pertinent Findings

No findings recorded

Biomarker Trends

No biomarker trends recorded for this dimension

⚪—

Nutritional HealthUnknown

No summary reasoning recorded.

Pertinent Findings

No findings recorded

Biomarker Trends

No biomarker trends recorded for this dimension

⚪—

Environmental HealthUnknown

No summary reasoning recorded.

Pertinent Findings

No findings recorded

Biomarker Trends

No biomarker trends recorded for this dimension

⚪—

Mental HealthUnknown

No summary reasoning recorded.

Pertinent Findings

No findings recorded

Biomarker Trends

No biomarker trends recorded for this dimension

⚪—

Physical ResilienceUnknown

No summary reasoning recorded.

Pertinent Findings

No findings recorded

Biomarker Trends

No biomarker trends recorded for this dimension

⚪—

Healthy LongevityUnknown

No summary reasoning recorded.

Pertinent Findings

No findings recorded

Biomarker Trends

No biomarker trends recorded for this dimension

⚪—

Men's HealthUnknown

No summary reasoning recorded.

Pertinent Findings

No findings recorded

Biomarker Trends

No biomarker trends recorded for this dimension

3c:T5677,

Microbiomics

Collected

July 16, 2026

Diversity

Diversity describes how many taxa are present and how evenly they are spread. Alpha diversity measures richness and evenness within this one sample, while beta diversity compares the whole community with a healthy reference population. Read together, they indicate whether a microbiome is thin, dominated by a few taxa, or simply distinct from the cohort.

Alpha diversity

Alpha diversity summarizes within-sample diversity through several complementary signals, each shown as a percentile relative to a healthy cohort. Shannon captures overall diversity, combining how many taxa are present with how evenly they are distributed. Richness counts the taxa observed. Berger–Parker dominance is the share of the community held by its single most abundant taxon. Higher values are more diverse for Shannon and Richness, whereas lower dominance indicates a community less concentrated in one taxon. Alpha diversity is not disease-specific and should be interpreted with other findings and clinical context.

Shannon

overall diversity

4.06

20th pct

LowAvgHigh

Richness

species observed

292

50th pct

LowAvgHigh

Dominance

Berger–Parker

14.9%

88th pct

LowAvgHigh

Most abundant: Bacteroides stercoris — 14.7% of the community

Beta diversity

Beta diversity compares the patient’s whole microbial community with a healthy reference cohort. The percentile reflects how far the composition sits from the cohort average: lower values are more typical, higher values more distinct — the opposite direction to alpha diversity. It reflects the whole community rather than the plotted axes, so it is the same in the 2D and 3D views. The plot is a PCoA projection where each point is one individual’s microbiome; points closer together are more similar in composition, and the patient is highlighted for comparison. PCoA is descriptive and the axes do not map directly to specific taxa or clinical outcomes.

View

Sex

Age

More distinct from the cohort average than 69% of healthy individuals (69th percentile)Rachel Delaney

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Most software asks you to work its way. Bioscope.ai applies your clinic's priorities and leaves clinical judgment with you.

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